Supplements: Dietary Nitrates — Beetroot Juice and Exercise Performance

Category: performance Updated: 2026-04-03

Beetroot juice at 400–600mg nitrate (70ml concentrate) reduces O2 cost at submaximal exercise by 1–3% and improves time trial performance by ~1–2%. Peak plasma nitrate occurs 2–3 hours post-ingestion. Elite athletes show consistently smaller responses than recreational athletes.

Key Data Points
MeasureValueUnitNotes
Evidence Tier2tierTier 2 — consistent mechanistic and performance evidence in recreational/trained athletes; attenuation in elite athletes reduces broad applicability
Standard Nitrate Dose400–600mg NO3−Per 70ml beetroot juice concentrate shot; standard single dose for acute performance studies
O2 Cost Reduction (submaximal)1–3%Nitric oxide reduces the O2 cost of producing a given mechanical output — effectively improving exercise efficiency
Time Trial Improvement~1–2%Across cycling and running time trials in trained recreational athletes; effects are smaller in elite athletes
Peak Plasma Nitrate Timing2–3hours post-ingestionPeak plasma NO3−/NO2− at approximately 2–3 hours after ingestion; consume 2–3h before exercise
Duration of Loading Protocol3–7daysChronic loading (daily doses for 3–7 days) may provide greater ergogenic effect than single acute dose
Oral Bacteria RequirementCriticalfactorNO3−→NO2− conversion requires oral bacteria; antibacterial mouthwash before beetroot juice eliminates most of the effect

The Nitrate Pathway: NO3− → NO2− → NO

Unlike most supplements where the mechanism is direct, dietary nitrate works through a two-step biological conversion pathway that depends critically on gut and oral bacteria:

  1. Absorption: Inorganic nitrate (NO3−) from food or supplements is absorbed primarily in the small intestine
  2. Salivary secretion: ~25% of absorbed NO3− is actively secreted into saliva by salivary glands
  3. Bacterial reduction: Oral bacteria reduce NO3− to nitrite (NO2−) — this step is eliminated by antibacterial mouthwash
  4. Gastric reduction: Swallowed NO2− is reduced to nitric oxide (NO) in the stomach (especially under acidic conditions)
  5. Systemic effects: NO causes smooth muscle relaxation (vasodilation), reduces the O2 cost of mitochondrial ATP production, and may improve contractile efficiency

The efficiency of step 3 is highly individual, explaining some of the variation in response to dietary nitrate supplementation.

Performance Response by Population

PopulationExercise TypeNitrate DosePerformance ImprovementEvidence QualityNotes
Untrained (<45 mL/kg/min VO2max)Cycling time trial400mg~3–5%Tier 2Largest responses seen
Recreationally trained (45–55 mL/kg/min)Running/cycling time trial400–600mg~1–3%Tier 1–2Majority of published RCTs
Well-trained (55–65 mL/kg/min)Cycling time trial400–600mg~0.5–2%Tier 2Smaller and more variable response
Elite (>65 mL/kg/min)Time trial400–800mg~0–1%Tier 2Hoon 2013: no significant effect in elite rowers
Older adults (60+)Submaximal cycling400mg~3–5%Tier 2Age-related decline in eNOS may increase response
Altitude exerciseCycling power output400–600mg~2–4%Tier 2Hypoxia enhances NO2−→NO conversion

Sources: Food vs Supplement

Concentrated beetroot juice shots (70ml) are the most extensively studied form, providing ~400–600mg NO3−. Dietary sources include:

  • Rocket/arugula: ~480mg NO3−/100g — most concentrated vegetable source
  • Spinach: ~250mg NO3−/100g
  • Beetroot (raw): ~250mg NO3−/100g
  • Celery: ~200mg NO3−/100g

Notably, food preparation matters: boiling vegetables removes 50–80% of nitrate into the cooking water. Raw or lightly steamed vegetables retain the most nitrate. Sodium nitrate supplements are also available but have less published research than the beetroot juice formulation.

What Doesn’t Work

Nitric oxide “booster” supplements based on L-arginine (the immediate NO precursor) have consistently failed to replicate the performance effects of dietary nitrate in healthy individuals. This is because intestinal first-pass metabolism and limited L-arginine uptake by eNOS in healthy endothelium makes oral L-arginine a poor NO substrate at typical supplement doses. The NO3−→NO pathway bypasses this limitation.

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Frequently Asked Questions

How does the nitrate-to-nitric oxide pathway work?

Dietary nitrate (NO3−) is absorbed in the small intestine and ~25% is secreted into saliva. Oral bacteria (primarily Veillonella and Actinomyces species) reduce salivary NO3− to nitrite (NO2−). Swallowed NO2− is absorbed and reduced to nitric oxide (NO) in the stomach and bloodstream — a process enhanced by low-oxygen (hypoxic) conditions such as exercising muscle. NO causes vasodilation (increases blood flow), improves mitochondrial efficiency, and reduces the O2 cost of ATP production.

Why do elite athletes respond less to dietary nitrates?

Elite athletes have chronically higher baseline nitric oxide production due to greater training volume, higher cardiac output, and more efficient endothelial NO synthase (eNOS) activity. When baseline NO production is already high, supplemental dietary nitrate has less additional effect — there is a ceiling on vasodilation and mitochondrial efficiency gains. Porcelli et al. (2016) showed the performance response is inversely correlated with VO2max: untrained athletes (~40 mL/kg/min) show the largest benefits; elite athletes (>65 mL/kg/min) show negligible effects.

Can mouthwash ruin the effect of beetroot juice?

Yes. Antibacterial mouthwash (especially chlorhexidine-based products) kills the oral bacteria responsible for NO3−→NO2− conversion. Studies show that using antibacterial mouthwash before or after beetroot juice consumption eliminates 60–90% of the salivary nitrite response and substantially attenuates blood pressure and performance effects. If using beetroot juice for performance, avoid antibacterial mouthwash on the day of supplementation. Standard toothpaste is less disruptive.

Is a single pre-exercise dose sufficient or is loading required?

Acute single doses (400–600mg, 2–3 hours before exercise) show significant performance effects in most studies. Chronic loading (same dose daily for 3–7 days) may produce greater plasma nitrite concentrations and potentially larger performance effects. For single-event competition use, an acute dose is practical. For training phases where nitrate's O2-efficiency benefits are targeted systematically, 3–7 days of loading before a key block is a reasonable protocol. Tolerance does not appear to develop with chronic use the way it does with caffeine.

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